Björn Vennström - Selected publications#


  • Wallis K, Dudazy S, van Hogerlinden M, Nordström K, Mittag J, Vennström B. The thyroid hormone receptor alpha1 protein is expressed in embryonic postmitotic neurons and persists in most adult neurons. Mol Endocrinol. 2010 Oct;24(10):1904-16.
  • Wallis, K., Sjögren, M., van Hogerlinden, M., Silbergberg, G., Fisahn, A., Nordström, K., Larsson, L., Westerblad, H., Morreale de Escobar, G., Shupliakov, O., Vennström, B. Aberrant development of subtype-specific GABAergic interneurons and locomotor deficiencies caused by an unliganded thyroid hormone receptor alpha 1. J Neurosci 28:1904-1915. (2008)
  • Sjögren, M., Alkemade, A., Mittag, J., Nordström, K., Katz, A., Rozell, B., Westerblad, H., Arner, A., Vennström, B. Hypermetabolism in mice caused by the central action of an unliganded thyroid hormone receptor alpha 1. EMBO J 26(21):4535-45. (2007)
  • Venero, C., Guadano-Ferraz, A., Herrero, A.I., Nordstrom, K., Manzano, J., de Escobar, G.M., Bernal, J., and Vennström, B. Anxiety, memory impairment, and locomotor dysfunction caused by a mutant thyroid hormone receptor alpha1 can be ameliorated by T3 treatment. Genes & Development 19(18): 2152-2163.(2005)
  • Tinnikov A, Nordstrom K, Thoren P, Kindblom JM, Malin S, Rozell B, Adams M, Rajanayagam O, Pettersson S, Ohlsson C, Chatterjee K, Vennstrom B. Retardation of post-natal development caused by a negatively acting thyroid hormone receptor alpha1. EMBO J. 2002 Oct 1;21(19):5079-87


Imprint Privacy policy « This page (revision-1) was last updated on Thursday, 6. December 2012, 15:47 by Nowak Aleksandra
  • operated by