Jürgen Knoblich - Publications#


Ten most relevant Publications:

Kraut, R., Chia, W., Jan, L.Y., Jan, Y.N., and Knoblich, J.A. (1996). Role of inscuteable in orienting asymmetric cell divisions in Drosophila. Nature 383, 50-55.

Schober, M., Schaefer, M., and Knoblich, J.A. (1999). Bazooka recruits Inscuteable to orient asymmetric cell divisions in Drosophila neuroblasts. Nature 402, 548-551.

Schaefer, M., Petronczki, M., Dorner, D., Forte, M. and Knoblich, J.A. (2001). Heterotrimeric G-Proteins Direct Two Modes of Asymmetric Cell Division in the Drosophila Nervous System. Cell, 107, 183-194.

Betschinger, J., Mechtler, K. and Knoblich, J.A. (2003). The Par complex directs asymmetric cell division by phosphorylating the cytoskeletal protein Lgl. Nature, 422, 326-330.

Emery, G., Hutterer, A., Berdnik, D., Mayer, B., Wirtz-Peitz, F., Gonzalez Gaitan, M., and Knoblich, J. A. (2005). Asymmetric rab11 endosomes regulate Delta recycling and specify cell fate in the Drosophila nervous system. Cell 122, 763-773.

Betschinger, J., Mechtler, K., and Knoblich, J. A. (2006). Asymmetric segregation of the tumor suppressor brat regulates self-renewal in Drosophila neural stem cells. Cell 124, 1241-1253.

Neumüller, R.A., Betschinger, J., Fischer, A., Bushati, N., Poernbacher, I., Mechtler, K., Stephen M. Cohen, S.M. and Knoblich, J.A. (2008). Mei-P26 regulates micro RNAs and cell growth in the Drosophila ovarian stem cell lineage, Nature, 454, 241-245.

Wirtz-Peitz, F., Nishimura, T., and Knoblich, J.A. (2008). Linking cell cycle to asymmetric division: Aurora-A phosphorylates the Par complex to regulate Numb localization, Cell, 135, 161-173.

Schwamborn, J.C. Berezikov, E., and Knoblich, J.A. (2009). The Brat homolog TRIM32 Prevents Self-renewal in Neural Progenitors by Degrading c-Myc and Activating Micro-RNAs, Cell, 136, 913-925.

Mummery-Widmer, J.L., Yamazaki, M., Stoeger, T., Novatchkova, M., Chen, D., Dietzl, G., Dickson, B.J., and Knoblich, J.A. (2009) Genome-wide analysis of Drosophila external sensory organ development by transgenic RNAi, Nature, 458, 987-992.
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