!!Rudolf Grosschedl - Selected publications
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__Total number of papers:__ 126\\
__H-factor:__ 68\\
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Giese, K., Cox, J., and Grosschedl, R. (1992).  The HMG domain of lymphoid enhancer factor 1 bends DNA and facilitates assembly of functional nucleoprotein structures.  Cell 69, 185-195. (615 citations)\\
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van Genderen, C., Okamura, R., Fariñas, I., Quo, R., Parslow, T., Bruhn, L., and Grosschedl, R. (1994).  Development of several organs that require inductive epithelial mesenchymal interactions is impaired in LEF-1 deficient mice.  Genes Dev. 8, 2691-2703. (745 citations)\\
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Lin, H., and Grosschedl, R. (1995).  Failure of early B cell differentiation in mice lacking the transcription factor EBF.  Nature 376, 263-267. (464 citations)\\
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Behrens, J., von Kries, J.P., Kühl, M., Bruhn, L., Wedlich, D., Grosschedl, R., and Birchmeier, W. (1996).  Functional interaction of β-catenin with the transcription factor LEF-1.  Nature 382, 638-642. (2272 citations)\\
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Jenuwein, T., Forrester, W.C., Fernandez-Herrero, L. A., Laible, G., Dull, M., and Grosschedl, R. (1997).  Extension of chromatin accessibility by nuclear matrix attachment regions.  Nature 385, 269-272. (243 citations)\\
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Sachdev, S., Bruhn, L., Sieber, H., Pichler, A., Melchior, F., and Grosschedl, R. (2001).  PIASy, a nuclear matrix-associated SUMO E3 ligase, represses LEF1 activity by sequestration into nuclear bodies.  Genes Dev. 15, 3088-3103. (470 citations)\\
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Dobreva, G., Chahrour, M., Dautzenberg, M., Chirivella, L., Kanzler, B., Farinas, I., Karsenty, G. and Grosschedl, R. (2006).  SATB2 is a multifunctional determinant of craniofacial patterning and osteoblast differentiation.  Cell 125, 971-986. (173 citations)\\
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Treiber, T., Mandel, E.M., Pott, S., Györy, I., Firner, S., Liu, E.T. and Grosschedl, R. (2010).  Early B Cell Factor 1 Regulates B Cell Gene Networks by Activation, Repression, and Transcription- Independent Poising of Chromatin.  Immunity 32, 714-725.\\
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Treiber, N., Treiber, T., Zocher, G., Grosschedl R. (2010).  Structure of an Ebf1:DNA complex reveals unusual DNA recognition and structural homology with Rel proteins.  Genes Dev. 24, 2270-2275. \\
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Nechanitzky, R., Akbas, D., Scherer, S., Györy, I., Hoyler, T., Ramamoorthy, S., Diefenbach, A., and Grosschedl, R. (2013).  Ebf1 is essential to maintain B cell identity and to prevent alternative cell fates in committed cells.  Nature Immunology 14, 867-875.